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Mil Lite 9 0 R2 FULL Version 43: The Best Subset of MIL X for Image Capture, Processing, and Analysis

teg studies are based on viscoelastic properties of whole blood [ 385, 386 ]. the basis of the teg methodology is the detection of changes in the viscosity and elasticity of the blood sample. different teg variables are correlated with the strength and the rate of fibrin formation. teg measurements are characterized by two different rheological parameters, the r-time (min) and the angle α. the r-time represents the time for the blood to form a clot. the angle α, that is the angle formed between the clot and the horizontal reference line in the tracing, represents the firmness of the clot. a shorter angle α results in a stiffer clot. a shorter r-time also indicates a stronger clot. the velocity vector (v) represents the velocity of the blood flow through the teg cup. the velocity vector is influenced by the arterial and venous blood flow. the maximum amplitude (ma) represents the height of the teg curve during clot formation. the ma is strongly influenced by the viscoelastic properties of the clot. the peak amplitude and the velocity vector do not have a pathophysiological meaning. teg has, however, proven useful as a diagnostic tool in various clinical conditions where coagulopathy is present [387-392].

Mil Lite 9 0 R2 FULL Version 43

activated protein c (apc) is a vitamin k-dependent, serine protease, which plays a pivotal role in the regulation of blood coagulation. apc inactivates coagulation factors va and viiia, which results in a slowdown of the coagulation cascade. the coagulation process is then further slowed by anticoagulant effects of the apc. apc is synthesized in the liver and circulates in plasma.

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